Alfa Mito Workshop Manual Elearn
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Category:2001 introductionsB-cell activation induced factor (BAFF), a member of the tumor necrosis factor (TNF) family, promotes the survival, maturation, and differentiation of B cells in a manner that is dependent on its ability to bind to its receptor, BAFF-R. BAFF is expressed in multiple tissues and is the best characterized cytokine known to be up-regulated in activated B cells.
Based on the structure of its receptor, BAFF has been found to play a key role in B-cell maturation, cell survival and homeostasis. BAFF mediates these activities by binding two ligands, BAFF-R and transmembrane activator and CAML interactor (TACI). Unlike BAFF-R, TACI does not have an intracellular domain. The interaction of BAFF with BAFF-R is relatively conserved and promotes TNF receptor associated factor 6 (TRAF6) recruitment, activation of NF-κB and induction of NF-κB-mediated gene expression. The binding of BAFF to TACI results in IL-6-like cytokine production by B cells.
There is increasing evidence that BAFF may play a role in the pathogenesis of autoimmune disease, including systemic lupus erythematosus (SLE) and rheumatoid arthritis (RA). Recent genetic association studies have also linked BAFF with multiple sclerosis.
There remains a need for effective modulators of BAFF.The protective effect of salidroside on doxorubicin-induced cardiotoxicity via the oxidative stress pathway.
Doxorubicin, an effective antimetabolite agent, is widely used for the treatment of malignancies. However, its clinical application is limited by cumulative cardiotoxicity. In the present study, we examined the protective effect of salidroside, the major active compound of Rhodiola rosea L., on doxorubicin-induced cardiotoxicity. Sprague-Dawley rats were randomly divided into five groups: a control group, a salidroside group and 3 doxorubicin treatment groups. Salidroside was administrated orally at doses of 50, 100 and 200 mg
Alfa Mito Workshop Manual Elearn
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